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It has been suggested that an increased EEC mass in response to chronic inflammatory injury drives neoplasia by producing trophic hormones (185)
Trial data showed significant drops in non-HDL cholesterol, triglycerides, and high-sensitivity C-reactive protein (a marker of cardiovascular inflammation)
Demographic and clinicopathologic characteristics were abstracted from medical records
Gut (2002) 51(1):3743.10.1136/gut.51.1.37 94 UysalKTWiesbrockSMMarinoMWHotamisligilGS
