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These divergent patterns of -arrestin utilization and endosomal signaling may underlie differences in drug-specific clinical profiles, including glucose control, durability of weight loss, and gastrointestinal tolerability
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Therefore, it is accepted that glucagon may affect energy expenditure via BAT-independent mechanisms (for review see [60, 61])
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9d), as also reported elsewhere in GLP-1R agonists administered peripherally in mice 49
