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This study found that GLP-1 treatment had a 2.11 HR of developing wet AMD: 0.2% of GLP-1 treated patients developed wet AMD compared to 0.1% of control

SGLT2is reduce oxidative stress by activating SIRT1/AMPK and inhibiting Akt/mTOR, thus collectively contributing to improved cellular energy efficiency, increased resistance to oxidative damage, and ultimately cardio- and renoprotection (reduced blood volume and blood pressure, promotion of diuresis and natriuresis, and glycosuria), especially beneficial in HF cases where excessive fluid can place additional strain on the heart [96,97]

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These molecules are administered mainly by sub-cutaneous injection, and as such, remain in the peripheral bloodstream for a long period of time (with half-lives of days), but have little access to the intestinal vagal afferents