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GLP-1 receptor activation can reduce neuroinflammation and oxidative stress, and restore insulin signaling, which may compensate for the dysfunctional dopaminergic neurotransmission and motor deficits associated with PD pathogenesis [74, 75]

The authors say many of these errors were preventable and could be reduced through better patient education

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J Clin Med 10(8):1798

AMPK and PGC-1 pathways also play an important role in autophagy in muscle during non-starvation status through the JNK/Bcl-2 anti-apoptotic pathway that dissociates the Beclin1/Bcl-2 complex and elicits autophagy [179]