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Molecular Pathways BPC-157 significantly promotes angiogenesis by enhancing vascular endothelial growth factor receptor-2 (VEGFR2) activity and nitric oxide (NO) signaling primarily through activation of the Akt-endothelial nitric oxide synthase (eNOS) pathway

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Semaglutide attenuates myocardial ischemia-reperfusion injury by inhibiting ferroptosis of cardiomyocytes via activation of PKC-S100A9 axis

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The journey from exendin-4 (the original Gila monster compound) to semaglutide took nearly two decades of clinical trials and refinement