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The weight maintaining effect of GLP-1RAs may be mediated by smaller decrease in free leptin and higher PYY336 response
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Conclusions The antifibrotic effects of GLP-1R activation on pulmonary fibrosis could be attributed to the inhibition of the interaction between NLRP3 inflammasome and PFKFB3-driven glycolysis, and histone lactylation in lung fibroblasts
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