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(13) These findings suggest a promising osteogenic role for GLP-1 therapies, especially in metabolic disease contexts

[2] Jin X, Qiu T, Li L, Yu R, Chen X, Li C, Proud CG, Jiang T (2023) Pathophysiology of obesity and its associated diseases

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1a) 52,57,58,59 , and truncation of a single C-terminal amino acid residue (L585) weakens FcRn binding affinity and, consequently, reduces its in vivo plasma half-life 60,65
