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Second, NFE2L2 activation enhances the expression of SLC7A11, GCLM, CBS, CHAC1, ABCC1/MRP1, GCLC and GSS, which are involved in GSH production, metabolism and release

[DOI] [PMC free article] [PubMed] [Google Scholar] 103.Pines A., Vrouwe M.G., Marteijn J.A., Typas D., Luijsterburg M.S., Cansoy M., Mullenders L

Nutritional Deficiencies Longitudinal melanonychia is seen in malnutrition especially protein energy malnutrition and vitamin D deficiency.[24] Vitamin B12 deficiency produces reversible nail pigmentation that can be longitudinal, diffuse bluish or reticulate probably due to reduced glutathione levels resulting in disinhibition of tyrosinase enzyme involved in melanogenesis.[25] Exogenous Pigmentation Brown to black pigmentation of nails can be seen due to exogenous agents like henna, dirt, tobacco, potassium permanganate, tar, and silver nitrate.[16] It presents as a horizontal band with the distal border parallel to the proximal nail fold and tends to move out with the nail plate [Figure 8].[16] Silver nitrate presents with a dark color band with irregular medial border and staining of adjacent skin of nail folds.[16] Syndromes Associated with Melanonychia LaugierHunziker syndrome, Peutz- Jegher and Touraine syndrome are characterized by LM along with pigmented macules in oral mucosa and lips.[7] LM involves several nails, generally finger nails with one or more bands

Nramp1 equips macrophages for efficient iron recycling

In T1DM, autoantibodies show specificity for binding two variants of ZnT8 (R or W at amino acid 325) dictated by a polymorphism in SLC30A8 [43]