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doi: 10.1016/j.neuroscience.2008.06.023
Dihydroergotoxine increased neuronal metabolism, and, in rats, it stimulated local glucose utilization in those parts of the brain related to learning and memory [142]
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All this indicates that an alteration of the barrier function in patients with CHF can induce translocations of bacteria and trigger the generation of cytokines, thus contributing to a deterioration of cardiac function [53]
[DOI] [PMC free article] [PubMed] [Google Scholar] 70.Denechaud P-D, Bossard P, Lobaccaro J-MA, Millatt L, Staels B, Girard J, Postic C
