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10.1056/NEJMra1905136 21 DeVivoD
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In contrast, mutations in VIT-1 and VIT-2, which are two major apolipoproteins for cholesterol trafficking between tissues, and in HEH-1, an ortholog of human NPC2, which is a soluble luminal protein in lysosomes and responsible for transferring cholesterol to human NPC1 (39, 58), hindered the effects of high cholesterol as ncr-1(0) (Fig 4D)
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