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We found that multiple molecules involved in lipid catabolism responded directly to glutamate deficiency as a presumed compensation for energy deficit

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superoxide radical, hydrogen peroxide, hydroxyl radicals, peroxyl radicals, nitric oxide, peroxynitrite), and their role in oxidative damage of DNA, proteins, and membrane lipids

This article explores key ligands that can enhance blood-brain barrier crossing of agar-based nanoparticles, including transferrin, lactoferrin, Angiopep-2, and glutathione, along with design strategies for improved brain delivery.