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Additionally, the regulatory properties of GST in kinase cascades have provided a translational opportunity to target GSTs in myeloproliferative pathways, with the consequent clinical testing of new agents in myelodysplastic syndrome

The only-defect group failed to regenerate during the experimental period, and the defect sites injected either nave or GSH-low hES-MSCs were partly filled with cartilage for 12 weeks (Fig