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In hepatocarcinogenesis, aflatoxin B1 (AFB1) exposure primarily induces oxidative stress through CYP3A4-mediated metabolic activation, generating ROS and MDA that directly cause DNA damage [389, 390]
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During the immune response, immune cells alter their metabolic activity (164), the metabolic reprogramming of DCs is essential for determining immunophenotypes and responses
[DOI] [PMC free article] [PubMed] [Google Scholar] 23.Grosse G.M., Schwedhelm E., Worthmann H., Choe C
Side effects are mild when medically supervised
