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We are aware that some of the subjects may have had genetically low concentrations of transcobalamin I (TCI), which is now known to be a benign condition that most probably does not need medical treatment [17]

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A search of the literature shows numerous other investigations regarding the role of the eNOS enzyme and NO itself, which finally suggest that NO in moderate amounts can be neuroprotective while NO in large concentrations will be neurotoxic or cytotoxic

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