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By incorporating miRNA structures into gene therapy agents, it aims to inhibit the production of the mutant Z-AAT protein and promote the production of functional A1AT ( These findings highlight the potential of A1AT augmentation therapy to modulate the inflammatory response in A1ATD, reduce neutrophil-mediated damage, and improve clinical outcomes in various manifestations of the disease, including lung inflammation and panniculitis

10.1128/AEM.01184-12 Appl

In conclusion, the pro-inflammatory state observed in aging populations may be related to dysfunction of the intestinal barrier

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