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doi: 10.1111/j.1528-1167.2012.03665.x 95
Similar to the mechanism of action of Rb1, Rd can significantly improve the systolic dysfunction, fibrosis, myocardial hypertrophy, inflammation and oxidative stress caused by pressure load in mice
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In cardiomyocytes, Rh2 reduced the levels of fibrotic signaling proteins, including signal sensor and activator of transcription 3 (STAT3), connective tissue growth factor (CCN2), and fibronectin
