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Consistently, GYY4137 treatment exhibited significant down-regulation of malondialdehyde content in LPS/ATP stimulated macrophages, which is commonly known as a marker of oxidative stress (Fig
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Intra-articular navitoclax injection on mice with post-traumatic OA effectively mitigated the proinflammatory microenvironment, restored subchondral bone damage, and enhanced cartilage maintenance
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