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The resultant oxidative stress adversely impacts cochlear homocysteine metabolism, suggesting a potential link between Vitamin B12 deficiency and demyelination of neurons in the cochlear nerve, axonal degeneration, and neuronal death

Some of the most convincing human data supporting the pore and leak pathways were generated in normal humans given a low pro-inflammatory intravenous dose of bacterial endotoxin and PEGs of MW 40010,000 Da

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