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These pathways are fundamental for neuronal survival and synaptic function, and their disruption has been associated with cognitive decline and neurodegeneration [182]

Importantly, the ingestion of the NOS inhibitor lNAME during 10 days of lowintensity muscle stimulation prevents faster to slower muscle fibre transition in rodents (Martins et al

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Key regulatory axesincluding HIF-1, HHIP-PKM2, PTGES-AKT-HIF-1, and lactate/LDH-A (LDH5)-TGF-interact to collectively promote enhanced glycolysis and diminished oxidative metabolism, sustaining the pathological phenotype of the disease (8082)