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Bevacizumab (Bev) is a monoclonal antibody that targets GBM angiogenesis by blocking VEGF signaling pathways that ultimately decrease blood supply to the tumor (39, 156158, 160)

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We, therefore, asked if S 0 could contribute to ferroptosis resistance by suppressing LPO in a GPX4-independent manner (Fig

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, Therefore, we used an RNAscope probe against Slc7a11 and revealed its expression within TLS (Figure 3B)

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doi: 10.1177/17557380231162873 70 AlbanoGD

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