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(101) found that adult 3-month-old SD male rats subjected to 3 months of EODF, compared to the normal diet group, retained glucose and glutamate uptake and mitochondrial function after synaptosomes were exposed to A protein damage, with increased stress protein levels, suggesting that EODF may alter synaptic homeostasis and enhance resistance to damage

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suggested that thiol-based catalysis is prone to overoxidation, whereas selenol-based catalysis is resistant to overoxidation [39]

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