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NMDAR hypofunction has been implicated in these alterations, with pre-clinical evidence that NMDAR disruption can lead to oxidative stress [33] and reduced PVI density in the hippocampus and PFC [34]
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Therefore, more alternative therapeutic strategies for AML patients are needed
A transcriptional program mediating entry into cellular quiescence
Fassett, D., Vaccaro A.R..: Classification Systems for Acute Thoracolumbar Trauma
