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Under ischemic conditions, dysfunctional mitochondria can promote cell death by increasing the release of pro-apoptotic factors, while mitophagy primarily functions to eliminate damaged mitochondria to maintain intracellular mitochondrial homeostasis [102]

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Here, we propose the hypothesis that a dysfunction during development in either NMDAR-mediated signaling, neuroimmune regulation, mitochondria function could initiate vicious circles centered on redox dysregulation/OxS, leading to persistent anomalies of PV neurons and oligodendrocytes and ultimately to neural synchronization, cognitive, emotional, social and sensory deficits characteristic of schizophrenia (Fig

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