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OxLDL polarizes macrophages towards the M1-like phenotype or M2-like phenotype depending on its extent of oxidation (323)
Overall, considering the evidence reported here, the cellular antioxidant defense in ALS appears to be strongly diminished, with an imbalance towards high and persistent OS levels
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CKD results in an altered equilibrium between activation of IGF-1 and SOCS, toward a markedly increased SOCS synthesis, which is most likely due to CKD-associated chronic inflammation resulting in a state of GH-insensitivity [156, 157]
