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Consequently, the oxidative balance is disturbed by the increased oxidative stress that, in turn, leads to DNA damage and contributes to accelerated cardiomyocyte death [86]

Although the mechanisms remain elusive, multiple studies over the last two decades have implicated skeletal muscle mitochondrial dysfunction in the development of insulin resistance (IR) [145,146,147,148,149,150]

van der Woude and Bumler, 2004

[159] work using a systematic amino acid rearrangement generated AMP with high proteolytic resistance with a membrane-disruptive mechanism in E

USP14 inhibition enhances Parkin-independent mitophagy in iNeurons