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The first step is the rate-limiting step and is driven by the enzyme glutamate cysteine ligase (GCL) (previously known as -glutamylcysteine synthetase and -glutamylcysteine synthase)

Alternatively, oral administration of NAC led to increased plasma GSH levels as well as effective penetration through the bloodbrain barrier to enhance GSH levels in the brains of animal models [13]

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there was no statistical significance between the two groups receiving different vitamin B12 concentrations

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