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In addition, we summarize the drivers of accelerated immunosenescence in PWH, including amplification of the senescence-associated secretory phenotype (SASP) arising from persistent low-grade inflammation and immune activation, as well as mitochondrial dysfunction jointly induced by HIV proteins (e.g., Vpr) and specific antiretroviral regimens (Fig

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Similarly, lowering iron levels in atherosclerotic tissue with the iron chelator Deferoxamine (DFO) could inhibit ferroptosis and inflammation, and mitigate the development of AS (Zhang et al., 2010)

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