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The induction of ferroptosis, combined with ER or AR antagonists, significantly inhibits the growth of ER-positive breast cancer or AR-positive prostate cancer, offering a new therapeutic approach for cancers with specific genetic background [48]

It protects cells from oxidative damage

Regulatory frameworks must evolve in tandem, embracing flexible, data-driven approaches that account for the complexity and subtlety of neuroprotective interventions

Based on the studies mentioned above, it is evident that copper overload can induce cell apoptosis through multiple molecular pathways, including the mitochondrial control pathway, endoplasmic reticulum stress-mediated pathway, and autophagy pathway