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This suggests that loss of SOD1 function, secondary to lack of copper metalation, contributes to the ALS phenotype
We also examined if the overexpression of ECM38 was able to suppress the disrupted vacuolar morphology of opt2 cells
GCs can increase sharply in response to environmental stressors to elicit physiological and behavioral adaptations by individuals to support survival (Sapolsky et al
Supplementary Figure 19
