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Targeting the TCA cycle through cuproptosis confers synthetic lethality on ARID1A-deficient hepatocellular carcinoma

Recent studies have demonstrated that in triple-negative breast cancer (TNBC), a substantial decrease in cancer cell GSH levels and a significant increase in cellular superoxide levels can be induced by the administration of a dual metabolic inhibitor of GLS and xCT (SLC7A11), which promotes apoptosis [46]

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