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Studies have shown that plasma TMAO may affect the course of various cardiovascular disorders and increase the risk of MI, despite the lack of direct relationships [65, 79]
doi: 10.1016/0003-9861(59)90516-8

2020;15:e0239506

Interestingly, evidence from metabolomic studies has shown associations between abnormal acylcarnitine and acetylcarnitine levels in several of the aforementioned fatigue-associated neuropsychiatric diseases, as reviewed elsewhere [29]

In vitro, ALCAR promoted the migration and proliferation of SC, and increased the expression of Pmp22 and NGF
